That condition and others like it are associated with an upper motor neuron lesion.

This reflex is observed in patients with upper motor neuron lesions.

The same mechanism applies for an upper motor neuron lesion in the right hemisphere.

It is one of the characteristic responses of an upper motor neuron lesion.

Hyper-reflexia (a response far larger than considered normal): Indicates a potential upper motor neuron lesion.

However in individuals with upper motor neuron lesions the jaw jerk reflex can be quite pronounced.

These findings are in contrast to findings in upper motor neuron lesions.

Following upper motor neuron lesions, affected muscles potentially have many features of altered performance including:

Upper motor neuron lesion (for example, hemiplegia the result of a cerebrovascular accident)

Hypertonia is caused by upper motor neuron lesions which may result from injury, disease, or conditions that involve damage to the central nervous system.

Historical progression of spasticity and the upper motor neuron lesion it is based on has progressed considerably in recent decades.

In upper motor neuron lesions, muscle tonus may increase and resistance of muscle to stretch increases.

Health professionals' understanding of impairments in muscles after an upper motor neuron lesion has progressed considerably in recent decades.

The Babinski sign can indicate upper motor neuron lesion constituting damage to the corticospinal tract.

Any lesion occurring within or affecting the corticobulbar tract is known as an upper motor neuron lesion.

Transneuronal degeneration of lower motor neurons is not present after upper motor neuron lesions in stroke patients.

The presence of the babinski sign after 12 months is the sign of a non-specific upper motor neuron lesion.

Cerebral palsy is a term referring to a non-progressive developmental neuromotor disorder with an upper motor neuron lesion origin.

The effectiveness of medications varies between individuals, and varies based on location of the upper motor neuron lesion (in the brain or the spinal cord).

This sign can appear due to an upper motor neuron lesion or various other conditions (including inborn errors of metabolism) which include spasticity as a symptom.

This is in contrast to an upper motor neuron lesion, which often presents with spastic paralysis - paralysis accompanied by severe hypertonia.

Other secondary changes such as loss of muscle fibres following acquired muscle weakness are likely to compound the weakness arising from the upper motor neuron lesion.

The upper motor neuron lesion in the brain impairs the ability of some nerve receptors in the spine to properly receive gamma amino butyric acid.

If a muscle has impaired function following an upper motor neuron lesion, other changes such as increased muscle stiffness are likely to affect the feeling of resistance to passive stretch.

Upper motor neuron syndrome (UMNS) is the motor control changes that can occur in skeletal muscle after an upper motor neuron lesion.