The best understood form of long-term potentiation is induced by the activation of the receptor complex.
Other receptor complexes are believed to be involved, in particular the excitatory glutamate system.
If co-repressors dominate the mix, the receptor complex may be unable to turn genes on or may have only a weak effect.
There appear to be at least three distinct receptor complexes that bind with high affinity to amylin.
The reverse process has not been demonstrated, suggesting that formation of this receptor complex is antigen-induced.
This receptor complex is thought to mediate the anxiolytic, sedative, and anticonvulsant actions of the benzodiazepines.
They primarily act as short-range inhibitory signals and signal through multimeric receptor complexes.
Like the benzodiazepines, they exert their effects by binding to and activating the benzodiazepine site of the receptor complex.
Cells are replete with equally stunning receptor complexes decorating their surfaces.
Engagement of this receptor complex will result in a series of signaling cascade, which lead to the production of several cytokines.
