It may also be an effect associated with cell proliferation.

This gene may play a role in cancer cell proliferation.

These conditions will then result in a selective pressure towards cancer cell proliferation.

Some hormones play a role in the development of cancer by promoting cell proliferation.

Stimulating cell proliferation, by itself, is normally not enough to cause cancer, though.

Cell proliferation is constantly being turned on and off in the target organs.

Quantitation of cell proliferation was conducted as described above for p53.

This results in several responses including cell proliferation and angiogenesis.

As a result, cellular transformation and cancer cell proliferation occurs.

The protein is also involved in cell proliferation, migration, and differentiation.

The greater the cell proliferation, the greater the chance for cancer cells to develop.

Several theories of the factors causing smooth muscle cell proliferation have been made.

It plays an essential role in T cell proliferation and activation.

In that case, AP1 is no longer expressed, and cell proliferation does not occur.

This preparation has been shown not to affect epithelial cell proliferation.

When this spirals out of control, it can cause uncontrolled cell proliferation.

Under normal physiological conditions, all cell proliferation is regulated by the restriction point.

Tumor cells have high metabolic demands due to the increase in cell proliferation.

They also cause cell proliferation in the stomach.

A wide range of studies has been conducted investigating the role in cell proliferation, differentiation, death, and survival.

Those are mutations of normal genes, which function by putting brakes on cell proliferation.

Interestingly, no genes associated with apoptosis or cell proliferation were identified.

Galectin-1 may act as an autocrine negative growth factor that regulates cell proliferation.

Neural stem cell proliferation declines as a consequence of aging.

There is a correlation between expression of egfl7, cell proliferation and micro-vessel density.