This form of wiring is known as long term potentiation.

They are important for Long-term potentiation, learning and memory.

This is likely the mechanism through which they mediate long term potentiation.

The theory was validated in part by the discovery of long-term potentiation.

This process of synaptic strengthening is known as long-term potentiation.

Aged rats lose their capacity both to learn new tasks and to show potentiation.

The result is an overall increase in potentiation.

These skills would not come until the later half of the 20th century, at about the same time as the discovery of long-term potentiation.

Could such potentiation be a form of neurophysiological memory?

In some cases, depression can be observed instead of potentiation following the tetanic stimulus.

Long term potentiation is the primary mechanism by which plasticity occurs.

Long-term potentiation in particular gets its name from the length of time which the increased synaptic strength remains.

It has been reported that there is a length-dependence of potentiation in skeletal muscle.

It also inhibited the induction of long term potentiation.

These two drugs also work together to induce long-term potentiation in neuronal populations.

Reelin is a crucial factor for long term potentiation.

But the problem was that long-term potentiation takes place in the pyramidal cells of the hippocampus.

It should be noted however, that the potentiation action of ibogaine may make this a very risky procedure.

Long term potentiation is shown to improve learning and memory by strengthening the communication between specific neurons.

In the potentiation of pentobarbitone sleep test, carbenoxolone significantly showed sedative effect.

Moreover, the potentiation produced by arachidonic acid is comparably slow to develop.

Long-term potentiation is the persistent increase in the strength of a chemical synapse that lasts from hours to days.

CREB is necessary for the late stage of long-term potentiation.

Potentiation, according to Epstein, both inherits the method of deconstruction and moves beyond it.

The mechanism for this action is thought to be through promoting the induction of long-term potentiation between synapses in the brain.