Evidence for this comes from the neuromuscular junction.

Nerves excite muscles in a similar way at the neuromuscular junction.

The neuromuscular junction is where a neuron activates a muscle to contract.

This axon then forms a neuromuscular junction with the effectors.

In mice that are deficient for either protein, the neuromuscular junction does not form.

Actions on the neuromuscular junction will result in prolonged muscle contraction.

By doing so, they increase the action potential threshold in the neuromuscular junction, eliciting paralytic behavior.

LRP4 plays an important role in the formation of neuromuscular junctions.

The space between the nerve terminal and the muscle cell is called the neuromuscular junction.

The area where the nerve contacts the muscle is called the neuromuscular junction, or the motor end plate.