The mechanism is not well characterized, but there is some evidence that it may involve mitochondrial function.
Some mitochondrial functions are performed only in specific types of cells.
Our results demonstrate that Rho 0derivative experience increased oxidative stress and has impaired mitochondrial function.
A defect in one of those genes could upset mitochondrial function.
Nutrient sensing in yeast controls stress defense, mitochondrial functions, Sir2, and others.
Upon the onset of a cellular energy crisis, mitochondrial function tends to decline.
Work on characterization of mitochondrial functions in other apicomplexans is ongoing.
We conclude that an increase in lipid peroxidation is associated with the impairment of mitochondrial function.
Heat stress has been shown to cause problems in mitochondrial functions and can result in oxidative damage.
Most genes needed for mitochondrial and plastid function are located in the nucleus.
