Hypotonic hyponatremia is categorized in 3 ways based on the patient's blood volume status.

Similarly, for both diuretics and dopamine, the volume status of the control and treatment groups must have been similar.

The first step in management is to assess glycemic, electrolyte, acid-base, and volume status.

Volume status should still be adequately evaluated.

However, if volume status is monitored closely, diuretics can be useful in the conversion to nonoliguria.

Thus, if this strategy is to be used, circulatory volume status and end-organ function must be carefully monitored.

In such patients meticulous monitoring of blood pressure and volume status is essential during the perioperative period [7].

Volume status may be monitored with the use of a central venous catheter to avoid over- or under-replacement of fluid.

The treatment of hyponatremia depends on the underlying cause and whether the patient's blood volume status is hypervolemic, euvolemic, or hypovolemic.

This should be gradual to avoid sudden changes in systemic volume status which can precipitate hepatic encephalopathy, renal failure and death.

These individuals may be sensitive to small shifts in their intravascular volume status, and are prone to develop volume overload (congestive heart failure).

When the plasma osmolarity is low, the extracellular fluid volume status may be in one of three states: low volume, normal volume, or high volume.

Still, the intravascular component is usually of primary interest, and volume status is sometimes used synonymously with intravascular volume status.

Nevertheless, conflicting opinions on the question of volume status in children with severe malaria can be satisfactorily resolved only through prospective randomized trials that include both fluid resuscitation and control groups.

Because it tends to increase or maintain cardiac output, it is sometimes used in anesthesia for emergency surgery when the patient's fluid volume status is unknown (e.g., from traffic accidents).

Ultrasound can be utilized to assess a persons intravascular volume status and response to intravenous fluid therapy by measuring the size and respiratory change in the diameter of the IVC.

Proposed mechanisms include stimulation of the erythrocyte sodium: potassium pump, alteration in sodium and volume status, effects on plasma renin, neurogenic mechanisms and an influence on the peripheral vasculature (Tannen, 1985).

Kokot was one of the first individuals to study abnormalities of volume regulating hormones and of volume status in acute renal failure, and one of the first to document abnormalities in humans with renal ischemia, particularly renal artery stenosis.

In medicine, intravascular volume status refers to the volume of blood in a patient's circulatory system, and is essentially the blood plasma component of the overall volume status of the body, which otherwise includes both intracellular fluid and extracellular fluid.