The vocal ligament begins to be present in children at about four years of age.

Reinke's space is a potential space between the vocal ligament and the overlying mucosa.

Fibroblasts have been found mostly aligned in the direction of the vocal ligament, along bundles of fibers.

Newborns have a uniform monolayered lamina propria, which appears loose with no vocal ligament.

The vocal ligaments (or inferior thyroarytenoid) are two strong bands enclosed within the vocal folds.

The intermediate and deep layers of LPs compose the vocal ligament which is responsible for strain in phonation.

The infant lamina propria is composed of only one layer, as compared to three in the adult, and there is no vocal ligament.

The exception to this would be if the vocal folds are not stretched, as stretching of the vocal ligaments abducts the vocal processes.

With the onset of puberty, both men and women's voices alter as the vocal ligaments become more defined and the laryngeal cartilages harden.

They have a three layer construction of an epithelium, vocal ligament, then muscle (vocalis muscle), which can shorten and bulge the folds.

These fibers run roughly parallel to the vocal fold edge and these two layers of the lamina propria comprise the vocal ligament.

The vocal process is the anterior angle of the base of the arytenoid cartilage, as it projects horizontally forward and gives attachment to the vocal ligament.

The macula flava in newborn vocal folds is important for the growth and development of the vocal ligament and layered structure of the vocal folds.

Transition from modal voice to falsetto occurs when each vocal cord's main body, or vocalis muscle, relaxes, enabling the cricothyroid muscles to stretch the vocal ligaments.

Interestingly, the results show that the vocal fold mucosae were hypoplastic, and rudimentary, and like newborns, did not have any vocal ligament, Reinke's space, or layered structure.

Within the ECM community of vocal ligament, fibrous proteins such as elastin and collagen are pivotal in maintaining the proper elastic biomechanical property of vocal fold.

These differences in newborn vocal fold composition would also be responsible for newborns inability to articulate sounds, besides the fact that their lamina propria is a uniform structure with no vocal ligament.

The normal strain level of vocal ligament ranges from 0-15% during phonation These fibrous proteins exhibit distribution variations spatially and temporally due to fibroblast turnover during tissue maturation and aging.

The lateral portions are thinner and lie close under the mucous membrane of the larynx; they extend from the superior border of the cricoid cartilage to the inferior margin of the vocal ligaments, with which they are continuous.

It is to the vestibular ligaments (vestibular folds once covered with mucus membrane) what the rima glottidis is to the vocal ligaments (vocal folds once covered with mucus membrane)s - the space formed when the folds are separated.

The lower and deeper fibers of the muscle can be differentiated as a triangular band which is inserted into the vocal process of the arytenoid cartilage, and into the adjacent portion of its anterior surface; it is termed the Vocalis, and lies parallel with the vocal ligament, to which it is adherent.