The striatum's subthalamic nuclei send control signals to the globus pallidus, which initiates and modulates motion.

The weaker signals from subthalamic nuclei thus cause reduced initiation and modulation of movement, resulting in the characteristic movements of the disorder.

These tonically inhibiting neurons thus decrease their inhibitory influence on the internal globus pallidus and the subthalamic nuclei.

These neurons in turn project to the internal segment of the globus pallidus and to the subthalamic nuclei which form a closed loop by projecting back to the internal globus pallidus.

The projections of the external globus pallidus to the subthalamic nuclei causes these neurons to increase their firing rate, since the globus pallidus neurons are inhibited by medium spiny neurons.

The subthalamic nuclei have excitatory projections to the internal globus pallidus thus causing the internal globus pallidus neurons to increase their inhibititory influence on SNpr and VTh.

It is particularly linked to the subthalamic nucleus by two-way connections.

The subthalamic nucleus is lenticular in form and of homogeneous aspect.

Another strong innervation is observed in the subthalamic nucleus.

The axons arrives at the lateral border of the subthalamic nucleus.

A different movement disorder, called hemiballismus, may result from damage restricted to the subthalamic nucleus.

Its major part is the subthalamic nucleus.

It sends nerve fibres to the subthalamic nucleus and putamen.

Those projecting to the subthalamic nucleus and nigra reticulata 52,6%.

As suggested by its name, the subthalamic nucleus is located ventral to the thalamus.

This observation caused scientists to believe that hemiballismus outside the subthalamic nucleus did not occur.

The subthalamic nucleus receives its main afference from the lateral pallidum.

The subthalamic nucleus essentially provides the excitement needed to drive the globus pallidus.

Subthalamic nucleus stimulation is a new application of the original deep brain stimulation technique.

The subthalamic nucleus and lateral pallidum are both fast-firing pacemakers.

Hemiballismus caused by lesions in the subthalamic nucleus is more severe than other forms of the disorder.

This suggests that the subthalamic nucleus is plastic enough to adapt to small amounts of damage in order to resume normal function.

It was within this book that Luys provided the first description of the structure that is today called the subthalamic nucleus.

The axons of the external globus pallidum go essentially to the subthalamic nucleus.

It is generally caused by damage to the subthalamic nucleus (STN).

The subthalamic nucleus is thus, in 84,2% of the cases, the target of lateral pallidal neurons.

The lateral pallidum receives a lot of striatal axons, the subthalamic nucleus not.

The axons of subthalamic nucleus neurons leave the nucleus dorsally.

Those nuclei are the motor thalamus, the globus pallidus, and the subthalamic nucleus.

These patients are subjected to stimulation of the globus pallidus internus, or the subthalamic nucleus.

The pseudobulbar crying could also be induced by stimulation in the region of the subthalamic nucleus of the brain.