The sodium-potassium exchange pump is relatively slow in operation.

These mediator proteins are not produced, and so stimulation of sodium-potassium exchange sites in the collection tubule does not occur.

Digitalis works by inhibiting sodium-potassium ATPase.

Cellular swelling may occur due to cellular hypoxia, which damages the sodium-potassium membrane pump; it is reversible when the cause is eliminated.

As calcium hydride is a relatively mild desiccant, it is safe compared with more reactive agents such as sodium metal or sodium-potassium alloy.

Sodium (in sodium cooled fast reactors) or sodium-potassium alloy NaK are frequently used; in special cases lithium can be employed.

Ludwig Moser developed a lead-free sodium-potassium glass that is more ecologically friendly than lead glass yet is extremely hard; it remains the basis of their products.

Normally, sodium-potassium pumps in the membrane of cells (in this case, cardiac myocytes) pump potassium ions in and sodium ions out.

Iodine uptake against a concentration gradient is mediated by a sodium-iodine symporter and is linked to a sodium-potassium ATPase.

NaK, or sodium-potassium alloy, an alloy of potassium (K) and sodium (Na), is usually liquid at room temperature.

An important example is the sodium-potassium exchanger (or Na/KATPase), wthe ionic concentration balance that maintains the cell potential.

The bicarbonate ion will stimulate an exchange of cellular H for Na, thus leading to stimulation of the sodium-potassium ATPase.

Ustalic acid is an inhibitor of the sodium-potassium pump (Na/K-ATPase), found in the plasma membrane of all animal cells.

This excess sodium in the kidneys that is destined for excretion (urination) can cause Hyponatremia (low sodium) and can lead to kaliuresis by increasing sodium-potassium exchange.

However, dopamine may increase urine output by yet a fourth mechanism, that of inhibition of sodium-potassium ATPase at the tubular epithelial cell level [ 53].

The sodium-potassium ATPase is an active transporter within the membrane that pumps potassium back into the cell and sodium outside of the cell, against their concentration gradients.

Metal alloys that are liquid at room temperature include NaK, a sodium-potassium metal alloy, galinstan, a fusible alloy liquid, and some amalgams (alloys involving mercury).

FBRs have been built cooled by liquid metals other than sodium-some early FBRs used mercury, other experimental reactors have used a sodium-potassium alloy called NaK.

Due to the low toxicity and low reactivity of its component metals, galinstan finds use as a replacement for many applications that previously employed toxic liquid mercury or reactive NaK (sodium-potassium alloy).

Such food staples have fundamentally altered several key nutritional characteristics of the human diet since the Paleolithic era, including glycemic load, fatty acid composition, macronutrient composition, micronutrient density, acid-base balance, sodium-potassium ratio, and fiber content.

Sodium-potassium ATPases redistribute K and Na ions until the membrane potential is back to its resting potential of around -70 millivolts, at which point the neuron is once again ready to transmit another action potential.

By increasing the delivery of sodium to the distal renal tubule, chlortalidone indirectly increases potassium excretion via the sodium-potassium exchange mechanism (i.e. apical ROMK/Na channels coupled with basolateral NKATPases).

However, based on publications from SiGNa's CEO and collaborators, we know that, in the past, they have absorbed sodium into silica by coating commercially available silica gel with a liquid sodium-potassium alloy (Na2K) to create a black powder.

Insulin (e.g. intravenous injection of 10-15 units of regular insulin along with 50 ml of 50% dextrose to prevent hypoglycemia) will lead to a shift of potassium ions into cells, secondary to increased activity of the sodium-potassium ATPase.

These experiments studied pigments, lipids, amino acids, cell wall synthesis, DNA synthesis, ribosomes, sodium-potassium membrane pumps, cell membrane characterization, glycolate metabolism, cyclic nucleotide metabolism, protein kinases, and a catalog of genes that were turned on by silicon.