reinnervation

Grafts may also be needed to allow for appropriate reinnervation.

This leads to possible reinnervation of the target cell or organ.

There is no need to develop specific prostheses for targeted reinnervation.

The appearance of discrete spectral components in a few patients is considered to reflect cardiac reinnervation.

Targeted reinnervation has an efferent and an afferent component.

The characteristic of the sensory reinnervation was quantified.

If proper connections to the appropriate receptors are not established, aberrant reinnervation may occur.

Targeted reinnervation does not require any implants.

This further hinders chances for regeneration and reinnervation.

This reinnervation may occur as early as 1 to 2 years after transplantation and is assumed to be of sympathetic origin.

A reduction in chronaxie occurs during reinnervation.

Targeted reinnervation enables an amputee to control motorized prosthetic devices and to regain sensory feedback.

After a CFNG procedure the first signs of reinnervation usually occur between 4 and 12 months.

An emerging variant of this technique is called targeted sensory reinnervation (TSR).

The goal of the latter is only to achieve fast reinnervation of the mimetic muscle to prevent irreversible atrophy.

Targeted reinnervation could also utilize implantable electrodes to record more localized signals from the target muscle, so that crosstalk can be further mitigated.

By means of nerve transfer, targeted reinnervation can also provide sensory feedback, which has not been achieved by any other form of prosthetics aforementioned.

The most striking feature of targeted reinnervation compared to traditional myoelectric prosthetics is its ability to provide multiple signals to control multiple functions simultaneously.

In 3 out of 36 leg muscles, reinnervation occurred spontaneously and chronaxie shortened to 0.1 ms, which is the value of normal innervated muscle.

Future studies will also need to investigate whether the ACH scaffolds allow for reinnervation of the target in vivo after a spinal cord injury.

Dr. Todd Kuiken of the RIC developed the procedure, which he calls "targeted muscle reinnervation."

Accelerating neuroregeneration and the reinnervation of a denervated target is critically important in order to reduce the possibility of permanent paralysis due to muscular atrophy.

This change may represent a change in innervation pattern that may include reinnervation by a different type of motor neuron or loss of multiple innervations.

The processes that occur in peripheral regeneration can be divided into the following major events: Wallerian degeneration, axon regeneration/growth, and nerve reinnervation.

Targeted sensory reinnervation is a method by which skin near or over the targeted muscle is denervated, then reinnervated with afferent fibers of the remaining hand nerves.