Pyridoxine deficiency is a very rare condition, even in developing countries.

Treatment of pyridoxine deficiency lies with replacing the missing vitamin.

As previously mentioned, isoniazid is associated with pyridoxine deficiency.

Treatment and prevention of pyridoxine deficiency.

Pyridoxine deficiency in children treated with isoniazid.

The main cause of pyridoxine deficiency is a lack of pyridoxine in the diet.

Vitamin B12 depletion (Pyridoxine deficiency) was reported to be associated with pyridoxine-dependent seizures.

A positive diagnosis test for pyridoxine deficiency can be ascertained by measuring erythrocyte levels of aspartate aminotransferase and transketolase in serum.

Pyridoxine is used for preventing and treating low levels of pyridoxine (pyridoxine deficiency) and the "tired blood" (anemia) that may result.

Pyridoxine deficiency (also known as B deficiency) is a paediatric disease due to a lack of pyridoxine (or vitamin B).

In patients receiving isoniazid, pyridoxine deficiency can lead to a sideroblastic anemia because pyridoxine is a required cofactor in heme synthesis.

High levels of kynurenic acid have been identified in human urine in certain metabolic disorders, such as marked pyridoxine deficiency and deficiency/absence of kynureninase.

Cracks, splits, or holes in your tongue signify an acute niacin (vitamin B-3) deficiency, a possible pyridoxine deficiency, or malabsorption of nutrients in general.

It has been speculated that the cause of seizures in pyridoxine deficiency lies with abnormalities in the normal ratio of glutamic acid to GABA.

Decarboxylation of glutamic acid to gamma-aminobutyric acid (GABA, a neurotransmitter) and transamination of glutamic acid to alpha-ketoglutaric acid are both impaired in animals when suffering from pyridoxine deficiency.