platelet adhesion

It is believed to work by increasing capillary endothelial resistance and promoting platelet adhesion.

These inhibitors are substances that prevent the clot formation by preventing platelet adhesion.

Differential effects of dietary supplementation with fish oil or soy lecithin on human platelet adhesion.

Promotion of human platelet adhesion and aggregation by a synthetic, triple-helical mini-collagen.

Alpha-tocopherol, an effective inhibitor of platelet adhesion.

It had previously been recommended for its effects on platelet adhesion and reduction of vascular damage caused by the heartworms.

The mechanism for the maintenance of this functional integrity is yet to be established, but presumably would involve platelet adhesion to the endothelium.

Proteins buildup on the surface and facilitate platelet adhesion and tissue growth in the propagation stage.

This promotes platelet adhesion and aggregation.

Heparin has been shown to suppress platelet adhesion, complement activation and protein adsorption.

Disorders of platelet adhesion or aggregation:

Early studies of platelet function in diabetes used in vitro tests of platelet adhesion.

Additionally, this same preparation, Pycnogenol, has been found to normalize platelet adhesion (aggregation) so as to facilitate normal blood flow.

Due to the possible action of proanthocyanidins on limiting platelet adhesion, grape seed extract may act as a blood-thinner, increasing clotting time.

Functions include: Platelet adhesion and activation - the most important platelet collagen receptor in terms of signaling.

The earliest finding is the platelet adhesion to collagen fibres followed by aggregation and formation of a platelet plug.

It has been shown that collagen, endothelial cover of the vessel, exposed after the injury plays as an agonist in platelet adhesion and its activation.

Rao GHR: Role of platelet adhesion and aggregation on thrombus formation.

Nonsteroidal anti-inflammatory drugs that affect platelet adhesion and damage gastric mucosa are contraindicated, especially if thrombocytopenia is present.

Transcriptional regulator ERG is required for platelet adhesion to the subendothelium and regulates hematopoiesis.

For instance, in the rabbit, doses of aspirin sufficient to inhibit prostacyclin production do not lead to platelet adhesion to intact endothelium (Dejana et al, 1980).

Similarly, prostacyclin inhibited platelet aggregation on rabbit subendothelium but only prevented platelet adhesion at higher doses (Higgs et al, 1978).

It arises from a qualitative or quantitative deficiency of von Willebrand factor (vWF), a multimeric protein that is required for platelet adhesion.

We know that endogenous NO from endothelial cells as well as organic nitrates and nitroprusside treatment mediate suppression of platelet adhesion and aggregation.

Platelet aggregation, or platelet-platelet stickiness, can be regarded as a special case of platelet adhesion and follows adhesion of platelets to injured surfaces.