oxygen toxicity

They called having an oxygen toxicity attack "getting a Pete".

The prevention of oxygen toxicity depends entirely on the setting.

Studies show that, in the long term, a robust recovery from most types of oxygen toxicity is possible.

The first sign of oxygen toxicity is usually a convulsion without warning.

This kind of hyperoxia can lead to oxygen toxicity.

Some also indicate oxygen toxicity exposure and water temperature.

Breathing at depth can cause nitrogen narcosis and oxygen toxicity.

Two British scientists suggested that it was oxygen toxicity that caused the disease.

They're limited to "no decompression" depths and carry a danger of oxygen toxicity.

Air breaks are taken between oxygen breathing to reduce the risk of oxygen toxicity.

Use of 100% oxygen is limited by oxygen toxicity at deeper depths.

Pulmonary oxygen toxicity is an entirely avoidable event while diving.

Furthermore, oxygen toxicity becomes a factor at high oxygen concentrations.

Oxygen toxicity has now become a rare occurrence other than when caused by equipment malfunction and human error.

Oxygen toxicity is managed by reducing the exposure to elevated oxygen levels.

This can lead to nitrogen narcosis, oxygen toxicity, or decompression sickness.

This mechanism is involved in nitrogen narcosis, oxygen toxicity and decompression sickness.

Convulsions due to acute oxygen toxicity may render a diver unconscious.

FiO is typically maintained below 0.5 even with mechanical ventilation, to avoid oxygen toxicity.

For oxygen sensitive strains, some strategies can be used to prevent oxygen toxicity in food products.

For more information, see oxygen toxicity.

Central nervous system oxygen toxicity was first described in this publication and is sometimes referred to as the "Paul Bert effect".

Other complications include diaphragm atrophy, decreased cardiac output, and oxygen toxicity.

The use of pure oxygen for accelerated decompression is limited by oxygen toxicity.

The effects of oxygen toxicity may be classified by the organs affected, producing three principal forms: