It can be associated with a lesion of an optic radiation.

More posteriorly, this becomes the optic radiation.

A distinctive feature of the optic radiations is that they split into two parts on each side:

Likewise, damage to the left posterior brain or left optic radiation can cause a loss of the right field of vision.

Homonymous hemianopia (often upper quadrantic): Calcarine cortex or optic radiation nearby.

From the lateral geniculate body, fibers of the optic radiation pass to the visual cortex in the occipital lobe of the brain.

The axons from the lateral geniculate nucleus, through the optic radiation, end in the primary visual cortex around the calcarine fissure.

Neurons of the LGN send their axons through the optic radiation, a direct pathway to the primary visual cortex.

Information leaving the LGN travels out on the optic radiations, which form part of the retrolenticular limb of the internal capsule.

The optic radiations, one on each side of the brain, carry information from the thalamic lateral geniculate nucleus to layer 4 of the visual cortex.

The information they carry is sent through the optic radiations to the visual cortices, possibly after editing and gating by visual cortex exerting top-down control.

He demonstrated that homonymous hemianopsia was caused by lesions in the occipital lobe and optic radiation as well as the optic tract.

Information from the light on each retina is taken to the occipital lobe via the optic nerve and optic radiation, where it is interpreted as vision.

Retinal sensors convey stimuli through the optic tracts to the lateral geniculate bodies, where optic radiations continue to the visual cortex.

The electrically-encoded visual information leaves the parvocellular cells via relay cells in the optic radiations, traveling to the primary visual cortex layer 4C-β.

These UBOs are typically found in the Cerebral peduncle, pons, midbrain, globus pallidus, thalamus, and optic radiations.

The koniocellular cells receive their input from bistratified retinal ganglion cells exiting the optic tract, and send their information via relay neurons in the optic radiation to the primary visual cortex.

The tracts project to a relay station in the midbrain called the lateral geniculate nucleus, part of the thalamus, and then to the visual cortex along a collection of nerve fibers called the optic radiations.

Sensory visual information is sent from the retina of the eye to the Lateral geniculate nucleus (LGN) in the Thalamus, which relays the visual information to the primary visual cortex by the fibers of the optic radiation.

The optic radiation (also known as the geniculo-calcarine tract or as the geniculostriate pathway) is a collection of axons from relay neurons in the lateral geniculate nucleus of the thalamus carrying visual information to the visual cortex (also called striate cortex) along the calcarine fissure.

Although the exact mechanisms that underlie regaining visual field functions through visual neuroplasticity and VRT are not yet fully known, the reorganization of the primary visual cortex is thought to make new connections and pathways in the optic radiation to the LGN to help regain visual field functions.