He died of motoneuron disease, a family spokeswoman said.

Upper motoneuron lesions to the face often cause paralysis.

Firing frequency is defined as the number of neuronal signals sent per second on one motoneuron.

The motoneuron sends an axon to a muscle.

A motor unit consists of one alpha motoneuron and all the muscle fibres it innervates.

Discovery of dendritic inhibition in central neurons (at the mammalian motoneuron).

Another set are the brain diseases of later life like Parkinson's, Huntington's, Alzheimer's and motoneuron disease.

The longest axon of a human motoneuron can be over a meter long, reaching from the base of the spine to the toes.

Axons of beta motor neurons (as alpha and gamma motoneuron) are myelinated.

However no direct vestibular neuron to medial rectus motoneuron pathway exists.

For example, the size of motoneurons usually reflects the size of the motor unit that is controlled by the motoneuron.

The alpha motoneuron communicates with acetylcholine receptors on the motor end plate of the effector muscle.

The preliminary contact formed between the motoneuron and the myotube generates synaptic transmission almost immediately, but the signal produced is very weak.

In this path, a "fast" motoneuron can convert any muscle fiber into a fast-twitch muscle fiber.

Rall W. (1959) Branching dendritic trees and motoneuron membrane resistivity.

Mutations in GLEI have been identified in families with foetal motoneuron disease.

The ability of a motoneuron to branch at the neuromuscular junction represents the pre-synaptic plasticity of the motor unit.

After about a week, a fully functional synapse is formed following several types of differentiation in both the post-synaptic muscle cell and the pre-synaptic motoneuron.

"The effects of an analgesic (biofreeze) on the motoneuron pool excitability of the soleus muscle."

There are two kinds of beta motoneuron (as gamma motoneuron) that include:

This decrease occurs as a result of increased excitability of the motoneuron and decreased activity of inhibitory pathways.

A decrease in peak-to-peak H-reflex amplitude suggests a decrease in motoneuron excitability.

Vacuolar degeneration is most prominent at the onset of the disease and precedes motoneuron death by two to three months [ 15 ] .

It is currently unclear how this system regulates its output to effect motoneuron bursting, which in turn is responsible for inspiratory muscle innervation.

This effects cause an increasing in pudendal motoneuron reflex discharge (PMRD) latency.