The medial longitudinal fasciculus carries information about the direction that the eyes should move.

Some of its fibers are, however, continued upward into the brain under the name of the medial longitudinal fasciculus.

For the cell in the midbrain, see Rostral interstitial nucleus of medial longitudinal fasciculus.

This tract also contributes to the make-up of the medial longitudinal fasciculus (MLF).

One-and-a-half syndrome is associated with damage to the paramedian pontine reticular formation and the medial longitudinal fasciculus.

The vestibulo-oculomotor fibers are the portion of the medial longitudinal fasciculus which ascends to the oculomotor nucleus from the vestibular nuclei.

In addition, by the medial longitudinal fasciculus and oculomotor nuclei, they activate the medial rectus muscles on the right eye.

The syndrome usually results from single unilateral lesion of the paramedian pontine reticular formation and the ipsilateral medial longitudinal fasciculus.

This is due to damage to both the motoneurons and interneurons projecting through the medial longitudinal fasciculus to the contralateral medial rectus neurons.

Sends uncrossed fibers to CN 3 and 4 via the medial longitudinal fasciculus (MLF)

The medial longitudinal fasciculus (MLF) is a pair of crossed fiber tracts (group of axons), one on each side of the brainstem.

The PPRF is located anterior and lateral to the medial longitudinal fasciculus (MLF).

The vertical gaze is controlled by the rostral interstitial nucleus of medial longitudinal fasciculus and the interstitial nucleus of Cajal.

Medial vestibulospinal fibers join with the ipsilateral and contralateral medial longitudinal fasciculus, and descend in the anterior funiculus of the spinal cord.

Although more rare than horizontal, one-and-a-half syndrome from damage to the paramedian pontine reticular formation and the medial longitudinal fasciculus can be shown to affect vertical gaze.

Medial vestibulo-spinal tract (medial, lateral, inferior, vestibular nuclei), bilateral projection via descending medial longitudinal fasciculus to cervical segments.

Some are probably derived from the posterior part of the thalamus and from the superior colliculus, whereas others are believed to be continued downward into the medial longitudinal fasciculus.

Moreover, lesions to the axonal tract of interneurons (in the medial longitudinal fasciculus) have been shown to disrupt conjugate eye movements through the paralysis of the contralateral eye.

The medial vestibulospinal tract projects bilaterally from the medial vestibular nucleus within the medial longitudinal fasciculus to the ventral horns in the upper cervical cord (C6 vertebra).

As its name would indicate, it is located near the Abducens nucleus, and it sends signals to it, which in turn affects the lateral rectus and the medial longitudinal fasciculus.

Cell group B6 is located in the floor of the fourth ventricle dorsal to, and between, the right and left medial longitudinal fasciculus of the pons in the primate and the rodent.

In special cases, where only one eye deviates and the other does not, this often indicates a lesion (or damage) of the medial longitudinal fasciculus (MLF) which is a brainstem nerve tract.

Structures affected by the infarct are the PPRF, nuclei of cranial nerves VI and VII, corticospinal tract, medial lemniscus, and the medial longitudinal fasciculus.

The middle layer consists of nerve cells and nerve fibers; fibers enter it from the parietal lobe through the external capsule, while others are said to connect it with the medial longitudinal fasciculus.

It does not innervate nuclei for nerves III, IV, and VI because these are mediated by cortical projections and yoked together by the MLF, medial longitudinal fasciculus.