As dramatic as these changes are, the fatty change is reversible.

Fatty change and alcoholic hepatitis with abstinence can be reversible.

Depending on the cause and severity of the lipid accumulation, fatty change is generally reversible.

In this early stage, fat accumulates in the liver cells around the central vein (fatty change).

Some alcoholics develop acute hepatitis as an inflammatory reaction to the cells affected by fatty change.

Fatty change, or steatosis is the accumulation of fatty acids in liver cells.

The fatty change subsides and is replaced by fibrosis (scarring) and some chronic inflammation.

Obstruction also causes centrilobular necrosis and peripheral lobule fatty change due to ischemia.

No doubt the retreat of fatty change and the shrinking effect of scar tissue is responsible for the over-all decrease in liver size.

Steatosis (also called fatty change, fatty degeneration or adipose degeneration)

In the liver, the enlargement of hepatocytes due to fatty change may compress adjacent bile canaliculi, leading to cholestasis.

The presence of micronodular cirrhosis, Mallory bodies and fatty change within a single biopsy are highly suggestive of alcoholic injury.

When mild, fatty change may have no effect on cell function but more severe fatty change may impair cellular function.

Chronic congestion in the centrilobular region of the liver leads to hypoxia and fatty changes of more peripheral hepatocytes, leading to what is known as nutmeg liver.

In cellular pathology, steatosis (also called fatty change, fatty degeneration or adipose degeneration) is the process describing the abnormal retention of lipids within a cell.

Although any organ system may be involved, Reye syndrome is primarily characterized by distinctive, fatty changes of the liver and sudden (acute) swelling of the brain (cerebral edema).

This is consistent with observed ATP depletion of exposed cells and histopathological findings of mitochondrial and cell swelling, glycogen depletion in liver cells and fatty change in liver, heart and kidney.

However, the tea has also been observed to cause a "mild non-dose dependent systematic toxicity" in various tissues throughout the body, "such as congestion, fatty changes, and necrosis in liver, blood vessels, kidney, lung and testis, but the brain, eyes, intestines and heart were essentially normal."