They found that nucleotide excision repair capability increased systematically with species longevity.

It is involved in base excision repair.

This process is known as nucleotide excision repair.

In general, the findings of these studies indicated a good correlation between nucleotide excision repair capacity and life span.

This block can only be relieved by a repair system called nucleotide excision repair.

Nucleotide excision repair is a more general mechanism for repair of lesions.

This complex functions in nucleotide excision repair.

AP sites can be formed by spontaneous depurination, but also occur as intermediates in base excision repair.

The related nucleotide excision repair pathway repairs bulky helix-distorting lesions.

Nucleotide excision repair is more complex in eukaryotes than prokaryotes, but the general principle is similar.

Most damage to bases in DNA is repaired by the base excision repair pathway.

Excision repair is a term applied to several DNA repair mechanisms.

DNA polymerase beta maintains genome integrity by participating in base excision repair.

XPG can be regarded as a protein involved in nucleotide excision repair .

Both mechanisms form a single-strand break, which is then repaired by either short-patch or long-patch base excision repair.

CPDs are readily repaired by nucleotide excision repair enzymes.

Base excision repair is the mechanism by which damaged bases in DNA are removed and replaced.

It initiates base excision repair by removing damaged bases to create abasic sites that are subsequently repaired.

It induces DNA lesions usually corrected by nucleotide excision repair.

However it is no longer working in humans and other placental mammals who instead rely on the less efficient nucleotide excision repair mechanism.

Nucleotidyl transferase is a component of the repair pathway for single nucleotide base excision repair.

As part of this complex, it facilitates nucleotide excision repair by unwinding DNA.

This is called base excision repair (BER).

This is important because the nucleotide excision repair pathway is a mechanism thought to be encoded by a caretaker gene.

Correction of this mutation involves the use of alkyladenine glycosylase (Aag) during base excision repair.