There is further integration in the anterior horn cells of the spinal cord.

The fatal childhood disorder is associated with loss of anterior horn cells and infantile death.

Anterior horn cells, motoneurons located in the spinal cord.

The hyperexcitable corticomotoneurons drive anterior horn cells into metabolic deficit, resulting in cell degeneration and death.

In addition to an abnormally small cerebellum and brainstem including the pons, there is a degeneration of the anterior horn cells.

The syndrome appears to involve the spinal cord: specifically, the anterior horn cells subserving the affected muscles are often damaged.

If this exocitotoxic process occurs rapidly, it results in a more rapid death of anterior horn cells resulting in lower motor neuron disease.

Because a different population of anterior horn cells is stimulated with each stimulation, each F wave have a slightly different shape, amplitude and latency.

In addition cortico spinal tract lesions do not cause anterograde transneuronal degeneration of spinal anterior horn cells.

In one case, a biopsy of an affected muscle "revealed scattered atrophic fibers, indicating lesions in the anterior horn cells of the spinal cord".

Jean-Martin Charcot encouraged him to study neurology, and together they demonstrated atrophy of anterior horn cells in the spinal cord in poliomyelitis in 1869.

Golgi I: neurons with long-projecting axonal processes; examples are pyramidal cells, Purkinje cells, and anterior horn cells.

Fibres from the lateral vestibular nucleus also pass via the vestibulospinal tract, to anterior horn cells at many levels in the spinal cord, in order to co-ordinate head and trunk movements.

The main feature distinguishing PCH type 1 from PCH type 2 is that anterior horn cells are spared in PCH type 2.

As for dogs, while about 30% of the pyramidal fibres reach the loin region of the spinal cord (Intumescencia lumbalis), those fibres always end on interneurons, never directly on anterior horn cells.

Poliovirus attacks specific neurons in the brainstem and the anterior horn cells of the spinal cord, generally resulting in the death of a substantial fraction of the motor neurons controlling skeletal muscles.

Betz cells send their axons down to the spinal cord via the corticospinal tract where in humans they synapse directly with anterior horn cells, which in turn synapse directly with their target muscles.

The condition is a result of a loss of anterior horn cells localized to lumbar and cervical regions of the spinal cord early in infancy, which in turn is caused by a mutation of the TRPV4 gene.

Pathological descriptions of BVVL include injury and depletion of 3rd-7th cranial nerves, loss of the spinal anterior horn cells, degeneration of Purkinje cells, as well as degeneration of the spinocerebellar and pyramidal tracts (Prahbu 2005).

Spinal polio, the most common form of paralytic poliomyelitis, results from viral invasion of the motor neurons of the anterior horn cells, or the ventral (front) grey matter section in the spinal column, which are responsible for movement of the muscles, including those of the trunk, limbs, and the intercostal muscles.