Patients with alcoholic pancreatitis may be less likely to respond.

They develop in about 10% of patients with acute alcoholic pancreatitis.

The effects of alcoholic pancreatitis can be managed, but not easily reversed.

It is applicable to both gallstone and alcoholic pancreatitis.

Alcoholic pancreatitis can result in severe abdominal pain and may progress to pancreatic cancer.

Limiting yourself to one or two alcoholic drinks per day may significantly lessen your chances of developing alcoholic pancreatitis.

Pancreatic pseydocyst formation is a complication of severe pancreatitis and most commonly follows alcoholic pancreatitis.

In this study we have used this technique to assess whether there are any longterm changes in pancreatic function after recovery from acute alcoholic pancreatitis.

The gastric acid secretory capacity in chronic alcoholic patients and those with alcoholic pancreatitis has been studied, with contradictory results reported by different studies.

It is well known that only a minority of alcoholics are afflicted with clinical pancreatitis and the issue of individual susceptibility to alcoholic pancreatitis has recently been reviewed.

Thus is seems that the amount of alcohol consumed may also have an effect on the severity of alcoholic pancreatitis in the total population, as well as in individual subjects.

Ten male patients in clinical remission after between one and six attacks of clinically acute alcoholic pancreatitis were studied between two and 29 months after the most recent attack.

Pancreatic enzyme synthesis, turnover, and secretion were measured in 10 patients in clinical remission who had had one or more (one to six) attacks of acute alcoholic pancreatitis.

The pathogenesis of alcoholic pancreatitis can be approached in two ways:(i) by studying factors influencing the individual susceptibility to the disease or (ii) by studying constant effects of ethanol on the pancreas.

Darryl McDaniels, a 28-year-old member of the rap group Run-DMC, recently told a rap magazine that he had been hospitalized for alcoholic pancreatitis, the result of years of drinking as many as eight 40-ounce bottles of malt liquor a day.

The low turnover rates may be ascribed to continuing pancreatic cell malfunction after recovery from acute alcoholic pancreatitis and suggest that the decreased amylase secretion rates are partly a consequence of impaired amylase synthesis and not simply because of loss of pancreatic tissue.

Furthermore, as the amount of alcohol consumed before pancreatitis correlates with the severity markers of the disease in patients with the first acute alcoholic pancreatitis episode, the correlation between the alcohol consumption in Finland and the occurrence of severe pancreatitis in the country was also studied.