RNA editing

RNA editing results in an alternative amino acid being translated.

RNA editing of this receptor occurs at 4 locations in the rat.

The number of seizures inversely correlates with the amount of rna editing.

Other RNA editing events are found in mammals, plants, bacteria and viruses.

A third possibility is regulation of RNA editing.

RNA editing is essential for the normal functioning of the plant's translation and respiration activity.

These two features distinguishes them from tumors where RNA editing does not occur.

Such comparisons should reveal cases of RNA editing.

Thus, RNA editing evolved more than once.

This is the first reported case that indicates that atopy could be associated with RNA editing.

An example Neumann-Held gives of this is RNA editing.

A stem loop hairpin structure mediates the RNA editing.

RNA editing was first discovered within the mitochondria of kinetoplastid protozoans, where it has been shown to be extensive.

It is thought that RNA editing may account for the wide variation in phenotype of this condition even among siblings.

The pre-mRNA of this protein is subject to RNA editing.

Activation kinetics are unaffected by RNA editing.

RNA editing is extensively studied in relation to infectious diseases, because the editing process alters viral function.

In eukaryotes, this may correspond with short pauses during transcription that allow appropriate RNA editing factors to bind.

RNA editing occurs in the cell nucleus and cytosol, as well as within mitochondria and plastids.

In fact, RNA editing can restore the functionality of tRNA molecules.

Studies suggest that the protein may also be involved in other RNA editing or RNA processing events.

The most extensively studied form of RNA editing involves the ADAR protein.

Perhaps as many as 16% of pre-miRNAs may be altered through nuclear RNA editing.

There is some evidence for RNA editing of human WT1 mRNA.

As with alternative splicing of the gene RNA editing increases the number of isoforms of this protein.