proteolipid protein , PLP (skrót)

Myelin basic protein has been shown to interact with Proteolipid protein 1.

Proteolipid protein 1 is a protein associated with Pelizaeus-Merzbacher disease.

Proteolipid protein 2 is a protein that in humans is encoded by the PLP2 gene.

Some of the proteins are myelin basic protein, myelin oligodendrocyte glycoprotein, and proteolipid protein.

The proteolipid protein (PLP) autoantigen is expressed in alternatively spliced forms, which exhibit tissue specific expression.

Possible targets of the immune response include myelin basic protein (MBP) and proteolipid protein (PLP).

Myelin proteolipid protein (PLP or lipophilin) is the major myelin protein from the central nervous system (CNS).

Following ablation of MRF the expression of myelin genes such as proteolipid protein, myelin basic protein, myelin-associated glycoprotein and myelin oligodendrocyte glycoprotein drops rapidly.

PMD is generally caused by a recessive mutation of the gene on the long arm of the X-chromosome (Xq21-22) that codes for a myelin protein called proteolipid protein 1 or PLP1.

These involve the hepatitis B virus mimicking the human proteolipid protein (myelin protein) and the Epstein-Barr virus mimicking anti-myelin oligodendrocyte glycoprotein (contributes to a ring of myelin around blood vessels).

Dr Jordi Folch-Pi, one of the founders of ASN, is known for his discovery of mild methods to extract biological lipids and for the discovery of myelin proteolipid protein.

As an example, Leone et al. have placed the transcription unit under the control of the regulatory sequences of the myelin proteolipid protein (PLP) gene, leading to induced removal of targeted gene sequences in oligodendrocytes and Schwann cells.