The deep tendon reflexes provide information on the integrity of the central and peripheral nervous system.

Pain sensation is affected predominantly and deep tendon reflexes are reduced.

The doctor may find that your deep tendon reflexes (knee jerks) do not work properly.

On examination the patients may have weakness, and loss of deep tendon reflexes (rarely increased or normal).

Another consequence is the depression of deep tendon reflexes, causing hyporeflexia.

Individuals also may experience muscle wasting and decreased or absent deep tendon reflexes.

A reflex hammer is a medical instrument used by physicians to test deep tendon reflexes.

Diminished deep tendon reflexes also may be noted.

Deep tendon reflexes and autonomic nervous system functions are also lost or diminished in affected areas.

As he checked the deep tendon reflexes, then the plantar responses, a suspicion began to take root in his mind.

As such, there is no mortality rate relating to the condition; however, loss of deep tendon reflexes is permanent and may progress over time.

Adie syndrome is tonic pupil plus absent deep tendon reflexes.

Deep tendon reflexes (knee and ankle jerk).

Monosynaptic reflexes, such as the deep tendon reflexes, are not restored until Phase 3.

Decreased or absent deep tendon reflexes: When a doctor taps the knee with his hammer little or nothing happens.

Lower motor neuron lesion: absent deep tendon reflexes.

Exaggerated deep tendon reflexes.

Grade 3: neck complaints plus neurological signs such as decreased deep tendon reflexes, weakness and sensory deficits.

Patient appears to be paralyzed although trace deep tendon reflexes elicited from both biceps and quadriceps symmetrically.

Spasticity is stiff or rigid muscles with exaggerated, deep tendon reflexes (for example, a knee-jerk reflex).

To test your deep tendon reflexes, your doctor will use a rubber hammer to tap firmly on the tendon.

Microcephaly, sensorineural hearing loss, diminished deep tendon reflexes, seizures, and cognitive impairment are also found in some affected individuals.

Upon neurological examination, the patient showed muscle atrophy, fasciculation in all limbs and decreased deep tendon reflexes.

Forty-five percent of patients had some neurologic abnormalities on exam, including weakness, paresthesias, and diminished deep tendon reflexes.

Features required for diagnosis are progressive weakness in legs and often arms and areflexia (the absence of deep tendon reflexes).