DNA fragmentation can occur under certain conditions in a few different cell types.

The main factors could be DNA fragmentation and caspase-3 activation.

DNA fragmentation is a secondary consequence, rather than an integral cause, of apoptosis.

The relative dose administered determines the extent to which DNA fragmentation occurs.

The degree of DNA fragmentation can predict outcomes for in vitro fertilization.

It is sometimes termed DNA fragmentation (this term is used for other procedures as well).

DNA fragmentation is a biochemical hallmark of apoptosis.

The terminal stage of cell differentiation is apoptosis, during which cells undergo DNA fragmentation and die.

Apoptotic DNA fragmentation is a key feature of apoptosis, a type of programmed cell death.

Spontaneous or accidental DNA fragmentation is fragmentation that gradually accumulates in a cell.

DNA fragmentation is the separation or breaking of DNA strands into pieces.

Its main units of measurement is DNA fragmentation index (DFI).

There are many methods to assess the DNA fragmentation caused by apoptosis and also many commercial kits are available.

In addition to its role as a waste-management endonuclease, it has been suggested to be one of the deoxyribonucleases responsible for DNA fragmentation during apoptosis.

This pore allows the exchange of monovalent ions, resulting in DNA fragmentation and eventually apoptosis.

To provide biochemical evidence that the form of cell death observed was apoptosis, assays for oligonucleosomal DNA fragmentation and caspase-3 activity were performed.

It was found that OROV causes apoptosis by DNA fragmentation.

(See also apoptotic DNA fragmentation.)

Necrosis, on the other hand, is usually characterized by random DNA fragmentation which forms a "smear" on agarose gels.

DNA fragmentation is often necessary prior to library construction or subcloning for DNA sequences.

The enzyme responsible for apoptotic DNA fragmentation is the Caspase-activated DNase.