Ultimately cardiac output can be reduced to very low levels.

To maintain cardiac output at reduced pressure, the heart rate must be increased.

The heart will not be able to stretch as far or develop the pressure required to increase the cardiac output.

Clinical measurement of cardiac output has been available since the 1970s.

A larger heart results in higher cardiac output, as more blood is pumped out with each beat.

The general effect is one of reduced cardiac output and increased strain on the heart.

Such a position is associated with decreased cardiac output in most pregnant women.

The heart may be working well otherwise, but it cannot produce a high enough cardiac output to keep up with this extra need.

Increases in blood sugar, breathing and cardiac output are all required.

Blood pressure depends on total peripheral resistance and cardiac output.

Cardiac output is the term that describes the amount of blood your heart pumps each minute.

This effect is coupled with decreased body temperature and cardiac output.

Cardiac output increases by about 50%, mostly during the first trimester.

The degree of change in temperature is directly proportional to the cardiac output.

Their utility results from an increased cardiac output by increasing the force of contraction.

Cardiac output increases during physical activity due to an increase in both the heart rate and stroke volume.

Under ordinary conditions, the colon receives between 10% and 35% of the total cardiac output.

This gradient may be increased by increases in the heart rate or cardiac output.

Blood pressure is dependent upon two factors: cardiac output and peripheral resistance.

Doctors think about cardiac output in terms of the following equation:

This limits the heart's ability to fill with blood, which can also further lessen cardiac output.

If the cardiac output is very low, this can damage organs, particularly the kidneys.

Despite their relatively small size, the kidneys receive approximately 20% of the cardiac output.

Dopamine is used in patients with shock or heart failure to increase cardiac output and blood pressure.

The renal circulation receives around 20% of the cardiac output.