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Cystinosis is the most common cause of Fanconi syndrome in children.
Low levels of phosphate in the blood due to a disease called Fanconi syndrome.
It is the first predictive test available for Fanconi Syndrome.
It should not be confused with Fanconi syndrome.
Fanconi's anemia is different from Fanconi syndrome, a rare kidney disorder.
Different diseases underly Fanconi syndrome; they can be inherited, congenital, or acquired.
Fanconi syndrome is a disorder affecting the kidneys.
Fanconi syndrome is treatable and organ damage is reduced if treatment begins early.
Lead poisoning also leads to Fanconi syndrome.
Fanconi syndrome, likely a misnomer, as this entity was not described by Fanconi.
With this test, it is possible to more accurately determine the probability of a dog carrying the gene for Fanconi Syndrome.
For example, those with cystinuria, cystinosis, and Fanconi syndrome may form stones composed of cystine.
Fanconi anemia is not related in any way to Fanconi syndrome, a rare kidney disorder.
Fanconi syndrome.
At present it cannot be predicted whether these basenjis are carriers or normal; however, it is unlikely that they will develop Fanconi syndrome.
Due to low reabsorption at proximal tubule (Fanconi syndrome)
Tenofovir can cause acute renal failure, Fanconi syndrome, proteinuria or tubular necrosis.
Infants affected by nephropathic cystinosis initially exhibit poor growth and particular kidney problems (sometimes called renal Fanconi syndrome).
This, in turn, may impair the function of these cells and may be the cause of antiviral induced Fanconi syndrome.
Other features of the generalized proximal tubular dysfunction of the Fanconi syndrome are:
The toxic effect of lead causes nephropathy and may cause Fanconi syndrome, in which the proximal tubular function of the kidney is impaired.
The use of stavudine, didenosine, abacavir, adefovir, cidofovir and tenofovir has been associated with Fanconi syndrome.
Fanconi syndrome usually presents between 4 and 8 years of age, but sometimes as early as 3 years or as late as 10 years.
The kidney dysfunction presents as Fanconi syndrome: Renal tubular acidosis, hypophosphatemia and aminoaciduria.
Light chains produce myriad effects which can manifest as the Fanconi syndrome (type II renal tubular acidosis).