feedback inhibition

Often, the first committed step is regulated by processes such as feedback inhibition and activation.

This is accomplished primarily through two different methods, feedback inhibition and transcriptional control.

This method is also found in bacteria, but feedback inhibition is more prevalent.

Cumulative feedback inhibition through end product metabolites; and 4.

Metabolic pathways are often regulated by feedback inhibition.

The first is feedback inhibition.

Anthranilate synthase is also regulated by feedback inhibition.

D: The regulation of proline biosynthesis can be dependent on the initial controlling step through negative feedback inhibition.

All the enzymes in this biosynthetic pathway are subject to regulation via feedback inhibition and/or repression at the genetic level.

There are conflicting reports as to whether negative feedback inhibition exists in humans and whether it is mediated by cholecystokinin.

This fusion thus provided a physical basis for the allosteric pattern of control called sequential feedback inhibition [ 11].

Negative feedback inhibition would therefore explain the greater sensitivity of amylase secretion for the detection of pancreatic disease.

This feedback inhibition is reversed once ATP binds.

Phosphorylation at Ser40 relieves feedback inhibition by the catecholamines dopamine, epinephrine, and norepinephrine.

Allosteric control occurs as feedback inhibition by palmitoyl-CoA and activation by citrate.

Each pair includes a large subunit, which is thought to be responsible for catalysis, and a small subunit for feedback inhibition.

Each one of these pathways is regulated in a similar fashion to the common pathway; with feedback inhibition on the first committed step of the pathway.

Valine performs feedback inhibition to inhibit the Acetohydroxy acid synthase used to combine the first two pyruvate molecules.

Pepsin also undergoes feedback inhibition; a product of protein digestion slows down the reaction by inhibiting pepsin.

These isoenzymes all have the ability to help regulate synthesis of DAHP by the method of feedback inhibition.

Normally, adrenal steroids then signal their presence to the brain to moderate ACTH levels (feedback inhibition).

Non-brain GS responds to end-product feedback inhibition, while brain GS does not.

Due to feedback inhibition, L-dopa results in a reduction in the endogenous formation of L-dopa, and so eventually becomes counterproductive.

Most of the pathways of amino acid biosynthesis are regulated by feedback inhibition, in which the committed step is allosterically inhibited by the final product.

It is plausible that some or all of these effects on Smo result from a feedback inhibition mechanism that targets the activated Smo receptor.