The invasion of tooth dentinal tubules by bacteria can be seen in the photograph opposite.

The three dimensional configuration of the dentinal tubules is genetically determined.

Resorption continues up the dentinal tubules into the tooth crown.

This interaction may be essential in the invasion of dentinal tubules by P. gingivalis.

The dental canaliculi (sometimes called dentinal tubules) are the blood supply of a tooth.

This diminishes the ability for dental caries to progress within the dentinal tubules.

It states that the flow of fluid in dentinal tubules trigger receptors within the tooth.

The lines are outlines of dentinal tubules, tiny structures that carry moisture through a tusk.

The smear layer prevented all the sealers from penetrating dentinal tubules.

Dentinal tubules are irregular and are bigger in diameter.

The second layer however occludes the dentinal tubules and strongly adheres to the canal walls.

The dentinal tubules pass the stimulus to odontoblastic layer of the pulp which in turns trigers the response.

After complete setting, the maximum penetration depth of the sealers into the dentinal tubules was examined in upper, middle, and lower levels.

It is believed to be caused by coalescing & looping of terminal portion of dentinal tubules.

It is believed that potassium ions diffuse along the dentinal tubules to inactivate intradental nerves.

As caries turn into dentin, the number of permeable dentinal tubules correlates with the degree of pain.

Once the decay passes through enamel, the dentinal tubules, which have passages to the nerve of the tooth, become exposed, resulting in a toothache.

The carious process continues through the dentinal tubules, which are responsible for the triangular patterns resulting from the progression of caries deep into the tooth.

General maintenance of the dentinal tubule and dentinal fluid (ion/protein content etc.)

Neumann's sheath: Dentinal sheath which form the walls of the dentinal tubules.

The physical occlusion of dentinal tubules results from both the hydroxylapatite layer and the residual NovaMin particles.

Fluids within dentinal tubules are believed to be the mechanism by which pain receptors are triggered within the pulp of the tooth.

In vivo, intra pair comparisons of bacterial invasion into dentinal tubules beneath ground, fractured and acid treated surfaces were made.

Olgart came to a conclusion that acid produced by microorganisms may dissolve the smear layer allowing bacteria to pass into dentinal tubules.

Some examples of acidic foods are fruits, fruit juices and wine whose acids can remove smear layers and open dentinal tubules.