For more than 15 years cTnI has been known as a reliable marker of cardiac muscle tissue injury.

After a heart attack, for example, large patches of cardiac muscle tissue end up dying as a result of temporary oxygen deprivation.

TnC in human cardiac muscle tissue is presented by an isoform typical for slow skeletal muscle.

The berries contain cardiogenic toxins which can have an immediate sedative effect on human cardiac muscle tissue, and are the most poisonous part of the plant.

The toxins can also have an immediate sedative effect on the cardiac muscle tissue possibly leading to cardiac arrest if introduced into the bloodstream.

Sonomicrometry crystals are most commonly implanted within skeletal or cardiac muscle tissue to track length changes during an activity (heartbeat, flapping a wing, chewing, etc.).

Human troponin I is presented in cardiac muscle tissue by a single isoform with molecular weight 23876 Da and it consists of 209 amino acid residues.

It is a 52kD protein that is a subunit of intermediate filaments in skeletal muscle tissue, smooth muscle tissue, and cardiac muscle tissue.

The impulses also pass to another region of specialized cardiac muscle tissue, a relay point called the atrioventricular node, located in the wall between the right atrium and the right ventricle.

Only one tissue-specific isoform of TnI is described for cardiac muscle tissue (cTnI), whereas the existence of several cardiac specific isoforms of TnT (cTnT) are described in the literature.