It is also being studied for use in avoiding reperfusion injury.

It is a reperfusion injury that appears after the release of the crushing pressure.

If and when the brain is reperfused, a number of factors lead to reperfusion injury.

The inflammatory response is partially responsible for the damage of reperfusion injury.

Reduction of body temperature also reduces the inflammation response and reperfusion injury.

Reperfusion injury is a primary concern in liver transplantation surgery.

Following restoration of normal blood flow, reperfusion injury may also contribute to the damage to the colon.

If medics try to get the blood flowing again after only a few hours, the result is reperfusion injury instead of amelioration.

As in other chronic ulcers, reperfusion injury damages tissue.

Arrhythmias may be another manifestation of myocardial reperfusion injury.

Allowing blood to flow back into the limb, for example by elevating it, is necessary but also contributes to reperfusion injury.

Xanthine oxidase inhibitors are being investigated for management of reperfusion injury.

Crush syndrome may also cause reperfusion injury when blood flow to decompressed muscle is suddenly restored.

This pathway allows ischaemic postconditioning that helps protect against reperfusion injury.

This is called reperfusion injury.

Intracellular sodium accumulation during ischemia as the substrate for reperfusion injury.

Protection by coenzyme Q10 of tissue reperfusion injury during abdominal aortic cross-clamping.

Ischemia results from the dysfunction and, combined with reperfusion injury, causes the tissue damage that leads to the wounds.

Reperfusion injury is classified as the damage that ensues after restoration of blood supply to ischemic tissue.

Pretreatment it reduced reperfusion injury in volunteers.

Controlled rewarming has been cited in the literature as beneficial in preventing reperfusion injury.

It is being investigated as a possible treatment for reperfusion injury and stroke, tinnitus, and bipolar disorder.

Nonetheless, current researchers now understand some of the major factors that lead to chronic wounds, among which are ischemia, reperfusion injury, and bacterial colonization.

This minimizes reperfusion injury in ischemic attacks.

Reperfusion injury plays a part in the brain's ischemic cascade, which is involved in stroke and brain trauma.