They found that nucleotide excision repair capability increased systematically with species longevity.

This process is known as nucleotide excision repair.

In general, the findings of these studies indicated a good correlation between nucleotide excision repair capacity and life span.

This block can only be relieved by a repair system called nucleotide excision repair.

Nucleotide excision repair is a more general mechanism for repair of lesions.

This complex functions in nucleotide excision repair.

The related nucleotide excision repair pathway repairs bulky helix-distorting lesions.

Nucleotide excision repair is more complex in eukaryotes than prokaryotes, but the general principle is similar.

XPG can be regarded as a protein involved in nucleotide excision repair .

CPDs are readily repaired by nucleotide excision repair enzymes.

It induces DNA lesions usually corrected by nucleotide excision repair.

However it is no longer working in humans and other placental mammals who instead rely on the less efficient nucleotide excision repair mechanism.

As part of this complex, it facilitates nucleotide excision repair by unwinding DNA.

This is important because the nucleotide excision repair pathway is a mechanism thought to be encoded by a caretaker gene.

Its comoutagenic effects may be explained by interference with base and nucleotide excision repair, eventually through interaction with zinc finger structures.

This gene encodes a component of the nucleotide excision repair (NER) pathway.

Nucleotide excision repair (NER), which repairs damage affecting 2 30 nucleotide-length strands.

This allows DNA repair mediated by endogenous mechanisms like the nucleotide excision repair system.

Though historical studies have shown inconsistent results, genetic variation or mutation to nucleotide excision repair genes can impact cancer risk by affecting repair efficacy.

ERCC2, or XPD is a protein involved in transcription-coupled nucleotide excision repair.

Dimers may be repaired by photoreactivation or nucleotide excision repair, but unrepaired dimers are mutagenic.

Many pathways are coordinated by the FA pathway for this including; nucleotide excision repair, translesion synthesis and homologous recombination.

XPC is involved in the recognition of bulky DNA adducts in nucleotide excision repair.

Next to a vital function in transcription initiation, TFIIH is also involved in nucleotide excision repair.